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Myxoviruses do not induce non-specific alterations in membrane permeability early on in infection

Archives of Virology
|January 1, 1983
PubMed

Insights

Myxovirus infections in cells do not cause the non-specific membrane permeability changes previously suggested. Nutrient uptake is sometimes reduced, but ion gradients remain stable, indicating specific cellular responses rather than general leakiness.

Area of Science:

  • Virology
  • Cell Biology
  • Membrane Transport

Background:

  • Myxoviruses are known to infect various cell types.
  • Previous studies suggested that virus infections might lead to non-specific increases in cell membrane permeability.
  • Understanding cellular changes during infection is crucial for antiviral strategies.

Purpose of the Study:

  • To investigate the permeability characteristics of cells infected with myxoviruses.
  • To determine if myxovirus infections cause non-specific membrane leakiness.
  • To analyze the effects of infection on nutrient uptake and ion gradients.

Main Methods:

  • Measuring the concentrative uptake of nutrients in infected cells.
  • Quantifying intracellular potassium ion (K+) concentration.
  • Assessing the maintenance of the sodium ion (Na+) gradient across the plasma membrane.

Main Results:

  • Cells infected with Sendai virus (BHK cells) and measles virus (Vero cells) showed no change in permeability.
  • Cells infected with Sendai, influenza, measles, or mumps viruses exhibited a decreased ability to concentrate nutrients.
  • Intracellular K+ concentration and the Na+ gradient remained unchanged in all infected cells studied.

Conclusions:

  • Myxovirus infections do not induce the non-specific increase in membrane permeability observed with haemolytic paramyxoviruses.
  • The observed changes in nutrient uptake are specific and not indicative of general membrane leakiness.
  • Cellular responses to myxoviruses involve specific alterations rather than a generalized loss of membrane integrity.

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