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[Allergic bronchopulmonary aspergillosis. Apropos of 30 cases]

Le Poumon Et Le Coeur
|January 1, 1983
PubMed

Insights

Allergic bronchopulmonary aspergillosis (ABPA) often develops years after chronic asthma onset. Diagnosis involves eosinophilia, elevated IgE, and hypersensitivity to Aspergillus fumigatus.

Area of Science:

  • Pulmonology
  • Allergy and Immunology
  • Infectious Diseases

Background:

  • Allergic bronchopulmonary aspergillosis (ABPA) is a hypersensitivity lung disease.
  • It primarily affects patients with chronic asthma, often developing decades after asthma onset.
  • Understanding ABPA's clinical and immunological features is crucial for diagnosis and management.

Purpose of the Study:

  • To analyze the clinical characteristics and immunological findings in patients with ABPA.
  • To investigate the relationship between ABPA and chronic asthma.
  • To explore potential factors contributing to Aspergillus fumigatus proliferation.

Main Methods:

  • Retrospective analysis of 30 ABPA cases treated between 1967 and 1981.
  • Review of patient histories, focusing on asthma duration and ABPA onset.
  • Evaluation of chest radiography findings, including infiltrates, atelectasis, and bronchiectasis.
  • Assessment of immunological markers: circulating eosinophilia, skin tests, and total and specific IgE levels.

Main Results:

  • ABPA developed in 28 of 30 asthma patients, with an average delay of 29 years after asthma onset.
  • Common radiographic findings included recurrent infiltrates (28 cases) and proximal bronchiectasis (16 cases).
  • Constant eosinophilia, positive skin tests (immediate and delayed), and elevated total IgE (600-9400 IU/ml) with specific IgE to Aspergillus were observed.

Conclusions:

  • ABPA presents with diverse clinical and immunological features, including eosinophilia and IgE-mediated hypersensitivity to Aspergillus fumigatus.
  • The long delay between asthma onset and ABPA suggests a complex, possibly localized, predisposition to fungal proliferation.
  • Further research into local factors may elucidate the pathogenesis of ABPA in susceptible individuals.

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