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Catecholaminergic influences on motion sickness.

J B Lucot

    Brain, Behavior and Evolution
    |January 1, 1983
    PubMed
    Summary

    Catecholamines may influence motion sickness. Dopamine and epinephrine might stimulate it, while norepinephrine may inhibit it, though more research is needed to confirm these roles.

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    Area of Science:

    • Neuroscience
    • Physiology

    Background:

    • Catecholamines are neurotransmitters with potential roles in various physiological processes.
    • Motion sickness is a complex condition affecting many individuals.
    • Understanding the neurochemical basis of motion sickness is crucial for developing effective treatments.

    Purpose of the Study:

    • To review the existing evidence for the involvement of catecholamines in motion sickness.
    • To elucidate the specific roles of dopamine, epinephrine, and norepinephrine in the pathophysiology of motion sickness.

    Main Methods:

    • Literature review of anatomical, pharmacological, and physiological studies.
    • Analysis of the evidence implicating catecholamine precursors and neurotransmitters in motion sickness.

    Main Results:

    • l-Dopa, a precursor to catecholamines, is implicated in motion sickness.
    • Dopamine and epinephrine show potential stimulatory roles, supported by some evidence.
    • Norepinephrine appears to have an inhibitory role in motion sickness, based on available data.

    Conclusions:

    • Evidence suggests catecholamines play a role in motion sickness.
    • Specific neurotransmitters like dopamine, epinephrine, and norepinephrine may differentially modulate motion sickness.
    • Critical information is lacking for a comprehensive understanding of these neurotransmitter roles.

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