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Sequential acquisition of R-plasmids in vivo by Salmonella typhimurium

Insights

Antibiotic resistance in Salmonella typhimurium evolved rapidly in a patient with acute renal failure, acquiring new plasmids and resistance markers. This highlights the dynamic nature of antimicrobial resistance in clinical settings.

Area of Science:

  • Medical Microbiology
  • Infectious Diseases
  • Antimicrobial Resistance

Background:

  • Salmonella typhimurium is a significant human pathogen.
  • Antimicrobial resistance is a growing global health concern.
  • Patients with compromised renal function may be at increased risk for complex infections.

Observation:

  • A chronic alcoholic patient in acute renal failure developed Salmonella typhimurium infection.
  • Initial Salmonella typhimurium isolate was resistant to trimethoprim and sulphamethoxazole, carrying an 18 Md plasmid.
  • Following ampicillin treatment, the infection persisted, with isolates acquiring ampicillin resistance and a 55 Md plasmid.
  • Further treatment led to chloramphenicol resistance and acquisition of a 62 Md plasmid in a subset of isolates.
  • Other Gram-negative bacteria (Escherichia coli, Klebsiella pneumoniae) isolated from the patient shared similar plasmid profiles.

Findings:

  • Salmonella typhimurium demonstrated rapid acquisition of multiple antibiotic resistance plasmids during treatment.
  • The patient's peritoneal dialysis fluid became a reservoir for resistant bacterial strains.
  • Evidence suggests potential plasmid transfer between different bacterial species within the patient.

Implications:

  • This case underscores the rapid evolution and dissemination of antimicrobial resistance in vivo.
  • It highlights the importance of careful antibiotic selection and monitoring in immunocompromised patients.
  • Understanding plasmid dynamics is crucial for managing multidrug-resistant infections.

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