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Anti-intermediate filament antibody in mice infected with Plasmodium berghei

Journal of Clinical & Laboratory Immunology
|March 1, 1984
PubMed

Insights

Infection with Plasmodium berghei in mice triggers the development of anti-intermediate filament antibodies (anti-IF). These antibodies, initially IgM then IgG, correlate with parasite levels during malaria.

Area of Science:

  • Immunology
  • Parasitology
  • Cell Biology

Background:

  • Malaria, caused by Plasmodium parasites, is a significant global health concern.
  • The host immune response to Plasmodium infection is complex and not fully understood.
  • Intermediate filaments (IF) are crucial intracellular structural components.

Purpose of the Study:

  • To investigate the development of anti-intermediate filament antibodies (anti-IF) during Plasmodium berghei infection in mice.
  • To determine the class and titre of anti-IF antibodies.
  • To explore the relationship between anti-IF antibody response and parasitaemia.

Main Methods:

  • CBA mice were infected with Plasmodium berghei.
  • Sera were tested for anti-IF reactivity using indirect immunofluorescence on HEp2 cells.
  • Anti-IF antibody classes (IgM, IgG) and titres were monitored over time.
  • Parasitaemia rates were assessed.

Main Results:

  • All infected mice developed anti-IF antibodies that reacted with IF in HEp2 cells.
  • Control mice showed no anti-IF reactivity.
  • Anti-IF antibodies of the IgM class were detectable by day 11, followed by IgG.
  • A progressive increase in anti-IF titre was observed, correlating with parasitaemia.

Conclusions:

  • Plasmodium berghei infection induces a specific anti-intermediate filament antibody response in mice.
  • The anti-IF antibody response is dynamic, involving both IgM and IgG, and is linked to parasite load.
  • This finding suggests a potential role for IF in the host-parasite interaction during malaria.

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