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[Hyperprolactinemic ovarian insufficiency].

H K Rjosk, D Berg, K von Werder

    Wiener Medizinische Wochenschrift (1946)
    |September 15, 1984
    PubMed
    Summary
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    Hyperprolactinemia causes 20% of menstrual disorders, often due to pituitary adenomas. Treatment for hyperprolactinemia is not always necessary unless other endocrine issues or pregnancy are concerns.

    Area of Science:

    • Endocrinology
    • Reproductive Medicine

    Background:

    • Hyperprolactinemia accounts for approximately 20% of menstrual cycle disorders.
    • Pituitary adenomas are a common cause of hyperprolactinemia, even when sellar changes are not evident.

    Purpose of the Study:

    • To investigate the causes and management of hyperprolactinemia in women with menstrual cycle disorders.
    • To evaluate the necessity of treatment for hyperprolactinemia.

    Main Methods:

    • Review of cases presenting with hyperprolactinemic ovarian insufficiency.
    • Analysis of imaging findings (sellar changes) and hormonal profiles.
    • Assessment of treatment outcomes for dopamine agonist therapy.

    Main Results:

    • While 30% of cases showed sellar changes indicative of space-occupying lesions, pituitary adenomas were suspected in the remaining cases.

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  • Treatment for hyperprolactinemia was deemed unnecessary in the absence of other endocrine dysfunctions or desire for pregnancy.
  • Gradual dose escalation of dopamine agonists can mitigate treatment side-effects.

    • Conclusions:

    • Hyperprolactinemia is a significant contributor to menstrual dysfunction, frequently linked to pituitary adenomas.
    • Treatment decisions for hyperprolactinemia should be individualized based on clinical presentation and patient goals.
    • Dopamine agonist therapy for hyperprolactinemia can be managed effectively with dose titration to minimize adverse effects.