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Mediators in the asthmatic child

Acta Paediatrica Hungarica
|January 1, 1984
PubMed

Insights

Prostaglandin levels are lower in children than adults, but this doesn't explain infant airflow resistance. Mediator release, like histamine, occurs in children, suggesting similar mechanisms across childhood.

Area of Science:

  • Pediatric Pulmonology
  • Allergy and Immunology
  • Biochemistry

Background:

  • Prostaglandins (PGF) are implicated in premature infant hyaline membrane disease.
  • Investigating prostaglandin E (PGE) levels during growth is crucial for understanding pediatric respiratory conditions.
  • Lower PGE levels in children compared to adults do not fully account for infant airflow resistance.

Purpose of the Study:

  • To investigate prostaglandin E (PGE) levels in children during growth.
  • To explore the role and release of inflammatory mediators in pediatric asthma.
  • To compare mediator release and response to allergic stimuli in children and infants.

Main Methods:

  • Measurement of prostaglandin levels during growth.
  • In vitro studies of histamine release from basophils upon exposure to allergenic extracts.
  • Cutaneous tests and respiratory challenges to assess mediator action indirectly.

Main Results:

  • Prostaglandin E (PGE) levels are lower in children than in adults.
  • Histamine release from basophils was observed in asthmatic children's blood mixed with allergens.
  • Mediator action was indirectly demonstrated in children (5-15 years) via tests, but not in infants.

Conclusions:

  • While PGE levels differ, they don't explain infant airflow resistance.
  • Histamine release is a key mediator mechanism in pediatric asthma.
  • Qualitatively similar mediator release mechanisms are likely present in children aged 5-15 years.

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