Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Acute ethylene glycol poisoning.

R DaRoza, R J Henning, I Sunshine

    Critical Care Medicine
    |November 1, 1984
    PubMed
    Summary

    Ethanol can protect against ethylene glycol poisoning by inhibiting its metabolism. Early diagnosis and treatment with ethanol and hemodialysis are crucial for preventing severe organ damage from antifreeze toxicity.

    Related Experiment Videos

    Related Concept Videos

    You might also read

    Related Articles

    Articles linked to this work by shared authors, journal, and citation graph.

    Sort by
    Same author

    Cocaine increases beta-myosin heavy-chain protein expression in cardiac myocytes.

    Journal of cardiovascular pharmacology and therapeutics·2001
    Same author

    Vasoactive intestinal peptide: cardiovascular effects.

    Cardiovascular research·2000
    Same author

    Coronary artery blood flow: physiologic and pathophysiologic regulation.

    Clinical cardiology·2000
    Same author

    Quality of proof.

    Journal of analytical toxicology·1999
    Same author

    Vagal nerve stimulation releases vasoactive intestinal peptide which significantly increases coronary artery blood flow.

    Cardiovascular research·1999
    Same author

    Vagal nerve stimulation during muscarinic and beta-adrenergic blockade causes significant coronary artery dilation.

    Journal of the autonomic nervous system·1998

    Area of Science:

    • Toxicology
    • Metabolic Medicine

    Background:

    • Ethylene glycol, a primary component of antifreeze, is metabolized into toxic byproducts like oxalate.
    • These metabolites can lead to severe metabolic acidosis and multi-organ damage, including to the central nervous system, lungs, and kidneys.