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Teratogenicity and embryotoxicity of titanocene dichloride in mice

Toxicology
|November 1, 1984
PubMed

Insights

Titanocene dichloride (TDC) causes significant birth defects, including cleft palate, in mouse fetuses when administered during critical developmental periods. This antitumor agent also exhibits embryotoxic effects, reducing live fetuses and fetal weight.

Area of Science:

  • Toxicology
  • Developmental Biology
  • Pharmacology

Background:

  • Titanocene dichloride (TDC) is an antitumor agent with potential applications in cancer therapy.
  • Understanding the developmental toxicity of therapeutic agents is crucial for risk assessment during pregnancy.

Purpose of the Study:

  • To investigate the teratogenic and embryotoxic effects of titanocene dichloride (TDC) in a mouse model.
  • To determine the impact of TDC administration on fetal development at different gestational stages.

Main Methods:

  • Pregnant mice received single doses of TDC (30 or 60 mg/kg) on specific days of gestation (8, 10, 12, 14, or 16).
  • Fetuses were examined on day 18 for external, internal, and skeletal malformations, as well as toxic effects.
  • Evaluated parameters included malformation incidence, litter size, fetal body weight, and skeletal ossification.

Main Results:

  • TDC administration on days 10 and 12 of gestation resulted in a high incidence of cleft palate (10-50% depending on dose).
  • Costal malformations were observed in some fetuses, but no other major malformations were noted.
  • Significant embryotoxic effects included reduced litter size, dose-dependent fetal weight reduction, and delayed skeletal ossification.

Conclusions:

  • Titanocene dichloride (TDC) is a potent teratogen, particularly affecting palate development when given during mid-gestation.
  • TDC exhibits significant embryotoxicity, impacting fetal growth and skeletal development.
  • These findings highlight the risks associated with TDC exposure during pregnancy and necessitate careful consideration in therapeutic use.

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