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Trimethoprim resistance plasmids

Annales De Microbiologie
|September 1, 1984
PubMed

Insights

Trimethoprim resistance in enterobacteria decreased when trimethoprim (Tp) was prioritized for urinary tract infections. Continued antimicrobial therapy correlated with persistent trimethoprim-resistant (Tp-R) strains, which resolved upon cessation of treatment.

Area of Science:

  • Medical Microbiology
  • Infectious Diseases
  • Antimicrobial Resistance

Background:

  • Enterobacteria carrying resistance plasmids pose a significant challenge in hospital settings.
  • Trimethoprim (Tp) resistance, often plasmid-mediated, impacts treatment efficacy for bacterial infections.

Purpose of the Study:

  • To investigate the prevalence of trimethoprim resistance in enterobacteria within a general hospital.
  • To assess the impact of changing treatment strategies on the incidence of trimethoprim resistance plasmids (R-plasmids).

Main Methods:

  • Survey of 320 patients in medical wards over a 6-month period.
  • Identification of enterobacteria harboring resistance plasmids conferring trimethoprim resistance.
  • Correlation of antimicrobial therapy patterns with the prevalence of Tp-resistant strains.

Main Results:

  • Initially, 5.6% of patients harbored enterobacteria with trimethoprim resistance plasmids (R-plasmids).
  • When trimethoprim (Tp) became the first-choice treatment for urinary tract infections, the incidence of Tp-R-plasmid strains decreased to 4%.
  • Persistent antimicrobial therapy was associated with the continued presence of Tp-R-plasmid strains in the gut.

Conclusions:

  • Strategic use of trimethoprim can reduce the prevalence of trimethoprim-resistant enterobacteria.
  • Antimicrobial stewardship is crucial, as continuous therapy promotes the persistence of resistant strains.
  • Discontinuation of antimicrobial therapy facilitates the clearance of trimethoprim-resistant organisms.

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