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Cardiac conduction defects in patients older than 60 years with aortic stenosis with and without mitral anular
Insights
Older adults with aortic stenosis (AS) and mitral annular calcium (MAC) have a higher prevalence of conduction defects. This finding highlights the importance of considering MAC in AS patients over 60.
Area of Science:
- Cardiology
- Cardiac Electrophysiology
- Geriatric Medicine
Background:
- Aortic stenosis (AS) is a common valvular heart disease in older adults.
- Conduction defects can complicate cardiac conditions and surgical interventions.
- Mitral annular calcium (MAC) is frequently observed in patients with AS.
Purpose of the Study:
- To investigate the prevalence of conduction defects in patients over 60 with AS.
- To determine if MAC is associated with an increased risk of conduction defects in this population.
Main Methods:
- Retrospective analysis of 51 patients over 60 undergoing aortic valve replacement for AS.
- Patients were categorized based on the presence or absence of mitral annular calcium (MAC).
- Prevalence of various conduction defects was compared between groups.
Main Results:
- 61% of patients with AS also had MAC.
- Conduction defects were significantly more prevalent in patients with MAC (58%) compared to those without MAC (25%).
- No significant differences in age, coronary artery disease, hypertension, or diabetes were noted between groups.
Conclusions:
- Patients over 60 with AS have a high prevalence of MAC.
- The presence of MAC in older adults with AS is associated with a higher incidence of conduction defects.
- Combined AS and MAC warrants careful electrophysiological evaluation.
Abstract:
The prevalence of conduction defects was investigated in 51 patients older than 60 years with aortic stenosis (AS) who underwent aortic valve replacement. Thirty one of the 51 patients, (61%) had associated mitral anular calcium (MAC). The mean age and prevalence of coronary artery disease, systemic hypertension and diabetes mellitus were similar in both groups. The prevalence of conduction defects (atrioventricular block, sinoatrial disease, bundle branch block, left anterior hemiblock or intraventricular conduction defect) was 18 of 31 (58%) in patients with MAC and 5 of 20 (25%) in patients without MAC (p less than 0.025). We conclude that patients older than 60 years with AS have a high prevalence of MAC, and that the prevalence of conduction defects is higher in patients older than 60 years with combined AS and MAC than in patients with AS without MAC.