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Lecithin-cholesterol acyltransferase activity in children in the early neonatal period

Biology of the Neonate
|January 1, 1984
PubMed

Insights

Plasma cholesterol esterification increases significantly after birth, with lecithin-cholesterol acyltransferase activity rising by 3 months. Fractional esterification rates in children align with adult levels, despite lower initial molar esterification rates.

Area of Science:

  • Biochemistry
  • Pediatric Medicine
  • Lipid Metabolism

Background:

  • Plasma cholesterol esterification is crucial for lipid transport and homeostasis.
  • Lecithin-cholesterol acyltransferase (LCAT) is the primary enzyme responsible for cholesterol esterification.
  • Understanding LCAT activity changes in newborns is vital for assessing early lipid metabolism development.

Purpose of the Study:

  • To investigate the developmental changes in plasma cholesterol esterification rate shortly after birth.
  • To determine the activity of lecithin-cholesterol acyltransferase (LCAT) in newborns at different ages.
  • To compare LCAT activity and esterification rates in infants with adult values.

Main Methods:

  • Radioassay method using a "common" substrate was employed.
  • Activity of lecithin-cholesterol acyltransferase (EC 2.3.1.43) was measured.
  • Molar esterification rate (MER) and fractional esterification rate were calculated in serum samples.

Main Results:

  • Molar esterification rate (MER) in umbilical cord serum was approximately half of that in 3-day-old and 3-month-old infants, and adults.
  • A positive correlation was observed between MER and plasma unesterified cholesterol concentration.
  • Fractional esterification rates in infants were comparable to those in healthy adults.

Conclusions:

  • Cholesterol esterification rate significantly increases in the first few days after birth.
  • LCAT activity matures rapidly, reaching adult levels within 3 months.
  • Infant lipid metabolism, specifically cholesterol esterification, shows efficient development post-birth.

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