[Disorders of acid-base equilibrium in ureterocolic implantation]

Different mechanisms responsible for acidosis following ureterocolic implantation into the continuity of the colon are reviewed. An acid urine is excreted by the kidneys in patients with acidosis, as in normal subjects. The acidity of the urine emitted into the colon stimulates bicarbonate secretion by the colic mucosa, and reabsorption of chloride. Acidosis following ureterocolic implantation could be related to bicarbonate loss in the stools, a renal acidosis mechanism not being involved as an acid urine is being excreted into the colon. An interesting point is that spontaneous regulation of this acidosis occurs, the bicarbonates secreted by the colon, as well as the ammonia, being secondarily reabsorbed during stagnation of the urine in contact with the colic mucosa. Oral administration of bicarbonates provokes alkalinization and suppression of loss of bicarbonates from the colon, with resulting correction of the acidosis.