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[Pathophysiology of thoracic injuries].

U Börner, G Hempelmann

    Unfallchirurgie
    |June 1, 1983
    PubMed
    Summary

    This review details the pathophysiology of chest injuries, focusing on "acute thorax." It explains key symptoms like hemorrhagic shock, low cardiac output, and hypoxia, along with acute respiratory distress syndrome (ARDS).

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    Area of Science:

    • Trauma Surgery
    • Thoracic Medicine
    • Critical Care

    Background:

    • Systematic reviews on thoracic injury pathophysiology are lacking.
    • Chest trauma encompasses diverse injury types and mechanisms.
    • The 'acute thorax' is a critical condition requiring prompt understanding.

    Purpose of the Study:

    • To systematically review the pathophysiology of thoracic injuries.
    • To elucidate the mechanisms behind the 'acute thorax'.
    • To describe cardinal symptoms and related conditions like ARDS.

    Main Methods:

    • Comprehensive literature search for studies on thoracic trauma pathophysiology.
    • Analysis of injury mechanisms and resulting physiological derangements.
    • Synthesis of information on shock states and hypoxia in chest trauma.

    Main Results:

    • Chest injuries can lead to hemorrhagic shock, venous congestion with low cardiac output, and hypoxia.
    • Acute Respiratory Distress Syndrome (ARDS) is a significant complication.
    • Understanding these pathophysiological pathways is crucial for effective management.

    Conclusions:

    • A thorough understanding of thoracic injury pathophysiology is essential for clinical practice.
    • Early recognition and management of shock and hypoxia improve outcomes.
    • Further research into specific injury mechanisms and therapeutic interventions is warranted.

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