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[Electron microscopic study of the effect of methicillin on phagocytized Staphylococcus aureus cells]

Antibiotiki
|July 1, 1981
PubMed

Insights

Methicillin alters Staphylococcus aureus morphology, affecting cell walls and cytoplasm. Higher doses cause cell death, with concentrations exceeding in vitro minimum inhibitory concentrations.

Area of Science:

  • Microbiology
  • Pharmacology
  • Cell Biology

Background:

  • Staphylococcus aureus is a common pathogen.
  • Methicillin is an antibiotic used to treat Staphylococcal infections.
  • Understanding antibiotic mechanisms of action is crucial for combating resistance.

Purpose of the Study:

  • To investigate the ultrastructural changes in Staphylococcus aureus cells phagocytized by mouse macrophages following methicillin administration.
  • To correlate methicillin dosage with observed morphological alterations in bacteria.
  • To compare in vivo methicillin concentrations with in vitro minimum inhibitory concentrations (MIC).

Main Methods:

  • Electron microscopy of ultrathin sections was used to examine phagocytized Staphylococcus aureus cells.
  • Mouse peritoneal macrophages were infected with Staphylococcus aureus and treated with varying doses of methicillin (2.5, 5, 25, 50 mg/kg).
  • Methicillin concentration in peritoneal exudate was measured and compared to in vitro MIC.

Main Results:

  • Low methicillin doses (2.5, 5 mg/kg) affected division septa and cell walls, leading to cell enlargement and cytoplasmic changes, but no lysis.
  • High methicillin doses (25, 50 mg/kg) induced more severe damage, including cell wall perforations and granular cytoplasm indicative of cell death.
  • In vivo methicillin concentrations in peritoneal exudate were 2-4 times higher than the in vitro MIC.

Conclusions:

  • Methicillin induces dose-dependent morphological changes in phagocytized Staphylococcus aureus.
  • The observed in vivo concentrations suggest methicillin efficacy against Staphylococcus aureus within the peritoneal cavity.
  • These findings provide ultrastructural insights into methicillin's bactericidal effects on Staphylococcus aureus.

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