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Published on: January 18, 2018
Cigarettes and accelerated hypertension
Insights
Regular smoking is significantly more common in patients with accelerated hypertension than benign hypertension. Smokers with accelerated hypertension face a higher risk of developing this severe hypertensive phase.
Area of Science:
- Cardiology
- Nephrology
- Epidemiology
Background:
- Accelerated hypertension is a severe form of high blood pressure.
- Risk factors for accelerated hypertension require further investigation.
- Understanding lifestyle factors like smoking and alcohol consumption is crucial.
Purpose of the Study:
- To compare smoking and drinking habits between patients with accelerated hypertension and benign hypertension.
- To investigate the association between smoking, alcohol consumption, and the progression to accelerated hypertension.
Main Methods:
- A case-control study comparing 58 patients with accelerated hypertension to 58 matched controls with benign hypertension.
- Data collected on smoking status, alcohol consumption, and serum creatinine levels.
- Patient mortality data was also analyzed.
Main Results:
- A significantly higher proportion of patients with accelerated hypertension were regular smokers (66%) compared to the control group (45%).
- No significant differences were found in alcohol consumption between the groups.
- Patients with accelerated hypertension had higher serum creatinine levels and a higher mortality rate.
Conclusions:
- Smoking is more prevalent in patients with accelerated hypertension compared to benign hypertension.
- Regular smoking appears to increase the likelihood of developing the accelerated phase of hypertension.
- Further research into the link between smoking and severe hypertension is warranted.
Abstract:
The smoking and drinking habits of 58 patients with accelerated hypertension were compared with those of a control group of 58 patients with benign hypertension, each individually matched for age, sex and date of presentation. Thirty-eight (66 percent) of the patients with accelerated hypertension were regular smokers compared to 26 (45 percent) of the control group. This excess of smokers was significant, but the average number of cigarettes smoked by smokers in the two groups was similar. The number of patients known to consume alcohol was the same in the two groups; and there were no significant differences in amount of alcohol drunk. Smoking and alcohol habits were not related. Thirty-six patients (62 percent) with accelerated hypertension had serum creatinine levels greater than 0.12 mmol/l compared with nine (16 percent) of the control group. Seventeen (29 percent) patients with accelerated hypertension were known to have died compared with five (9 percent) of the control group. This survey confirms that, as shown by recent studies in Britain, smoking is more common in patients presenting with accelerated than with benign hypertension. It appears that hypertensive patients who smoke regularly are more likely to develop the accelerated phase than those who do not.
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