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[Identification and characteristics of the conjugative R plasmids of conditionally pathogenic E. coli isolated from

Antibiotiki
|June 1, 1980
PubMed

Insights

Most E. coli strains causing chronic pyelonephritis exhibit antibiotic resistance due to R-plasmids. These transferable plasmids, often F-like, carry multiple resistance markers, though unique DNA patterns were observed among them.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Antibiotic resistance is a growing concern in treating bacterial infections.
  • Conditionally pathogenic Escherichia coli (E. coli) strains can cause severe infections like chronic pyelonephritis.
  • R-plasmids are key genetic elements responsible for the spread of antibiotic resistance.

Purpose of the Study:

  • To investigate the prevalence of antibiotic resistance in E. coli strains from chronic pyelonephritis patients.
  • To characterize the R-plasmids present in resistant E. coli strains.
  • To analyze the genetic relatedness and diversity of these R-plasmids.

Main Methods:

  • Isolation and antibiotic susceptibility testing of E. coli strains.
  • Conjugation experiments to transfer R-plasmids.
  • Plasmid DNA extraction and restriction endonuclease digestion (EcoRI).
  • Agarose gel electrophoresis for fragment analysis.

Main Results:

  • 74.6% of 63 E. coli strains showed resistance to one or more antibiotics.
  • 7 strains harbored transferable R-plasmids, mostly F-like, with common antibiotic resistance markers.
  • Restriction analysis revealed that the studied R-plasmids were not identical.
  • Evidence suggests the presence of plasmid complexes with similar resistance markers within single strains.

Conclusions:

  • Conditionally pathogenic E. coli strains from chronic pyelonephritis patients frequently exhibit multidrug resistance.
  • Transferable R-plasmids play a significant role in disseminating antibiotic resistance.
  • Genetic analysis of R-plasmids is crucial for understanding resistance mechanisms and evolution.
  • The findings highlight the complexity of antibiotic resistance gene acquisition and maintenance in bacterial populations.

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