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Donor pretreatment on perfused cadaver kidneys: experimental observations

Related Concept Videos

Exogenous and endogenous nitric oxide but not iNOS inhibition improves function and survival of ischemically injured livers.

Neutrophil depletion and chemokine response after liver ischemia and reperfusion.

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Nitric oxide diminishes apoptosis and p53 gene expression after renal ischemia and reperfusion injury.

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