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Relationship between pigment producibility and drug resistance in Serratia marcescens

Insights

R plasmid transfer in Serratia marcescens can lead to non-pigmented, drug-resistant strains. This suggests R plasmid acquisition may explain the rise of these variants in clinical settings.

Area of Science:

  • Microbiology
  • Genetics
  • Molecular Biology

Background:

  • Serratia marcescens clinical isolates exhibit variations in pigmentation and drug resistance.
  • Non-pigmented variants are observed more frequently in drug-resistant strains.

Purpose of the Study:

  • To investigate the role of R plasmid transfer in the emergence of non-pigmented, drug-resistant Serratia marcescens.
  • To compare the frequency of R plasmid transfer between different strains and sources.

Main Methods:

  • Experimental transfer of R plasmid from Escherichia coli to pigmented Serratia marcescens strains.
  • Assessment of pigment production and drug resistance in recipient strains post-plasmid transfer.
  • Comparison of R plasmid recipient capabilities between pigmented and non-pigmented strains.

Main Results:

  • Transfer of R plasmid induced spontaneous loss of pigment production in pigmented strains.
  • Non-pigmented strains demonstrated higher efficiency in receiving R plasmids compared to pigmented strains.
  • R plasmid transfer occurred at a higher frequency from non-pigmented strains to pigmented strains than from E. coli.

Conclusions:

  • R plasmid transfer is a significant factor contributing to the increased prevalence of non-pigmented, drug-resistant Serratia marcescens.
  • The interaction between R plasmids and host cell genetics influences phenotypic changes like pigment loss.

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