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Neurogenic pulmonary edema

I A Fein, E C Rackow

    Chest
    |March 1, 1982
    PubMed
    Summary
    This summary is machine-generated.

    Neurogenic pulmonary edema following subarachnoid hemorrhage can occur due to increased capillary permeability, not necessarily altered pulmonary hemodynamics. This finding offers new insights into managing this critical complication.

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    Area of Science:

    • Neurology
    • Pulmonology
    • Critical Care Medicine

    Background:

    • Acute subarachnoid hemorrhage is a serious neurological event.
    • Pulmonary edema is a potential, life-threatening complication of subarachnoid hemorrhage.
    • The exact mechanisms underlying neurogenic pulmonary edema remain under investigation.

    Observation:

    • A patient developed fulminant pulmonary edema after an acute subarachnoid hemorrhage.
    • Hemodynamic assessment showed low-normal pulmonary arteriolar resistances.
    • Endobronchial fluid analysis revealed colloid osmotic pressure and protein content similar to the patient's plasma.

    Findings:

    • Neurogenic pulmonary edema in this case was associated with increased pulmonary capillary permeability.
    • The edema occurred despite normal pulmonary hemodynamics, suggesting an alternative mechanism.

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  • Pulmonary fluid composition mirrored plasma, indicating a non-cardiogenic origin.

    • Implications:

    • This case suggests neurogenic pulmonary edema can arise from capillary leak syndrome independent of hemodynamic changes.
    • Understanding this mechanism may guide therapeutic strategies for post-subarachnoid hemorrhage pulmonary edema.
    • Further research is warranted to explore the role of capillary permeability in neurogenic pulmonary edema.