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Objective evidence of occult myocardial dysfunction in patients with frequent ventricular ectopy without clinically

Insights

Frequent ventricular ectopic activity (VEA) in asymptomatic individuals may indicate subclinical myocardial dysfunction. These findings suggest subtle left ventricular abnormalities even without apparent heart disease.

Area of Science:

  • Cardiology
  • Electrophysiology
  • Cardiovascular Imaging

Background:

  • Frequent ventricular ectopic activity (VEA) is often detected incidentally.
  • The association between VEA and subclinical cardiac dysfunction in asymptomatic individuals requires further investigation.

Purpose of the Study:

  • To investigate left ventricular (LV) function in asymptomatic individuals with frequent VEA and normal coronary arteriograms.
  • To identify potential subclinical myocardial dysfunction associated with frequent and complex VEA.

Main Methods:

  • Inclusion of 18 asymptomatic individuals with frequent VEA (>100 b/hr) and normal coronary arteriograms.
  • Assessment using 24-hour ambulatory ECG, cardiac catheterization, LV angiography, and hemodynamic measurements.
  • Evaluation of LV volumes, end-diastolic pressure, ejection fraction, and myocardial contractility (mean velocity of circumferential fiber shortening).

Main Results:

  • 13 individuals (72%) had complex VEA; 8 had undiagnosed hypertension.
  • Elevated LV end-systolic volume index (56%), LV end-diastolic volume index (67%), and LV end-diastolic pressure (61%) were observed.
  • Impaired myocardial contractility (decreased mean velocity of circumferential fiber shortening <1.0 circ/sec) was found in 56% of individuals.
  • LV dysfunction was more prevalent with higher VEA frequencies (>300 b/hr).

Conclusions:

  • Subclinical myocardial dysfunction is present in some individuals with frequent VEA, characterized by subtle LV volume and pressure abnormalities.
  • Decreased mean velocity of myocardial circumferential fiber shortening indicates impaired contractility.
  • The etiologic mechanisms for frequent and complex VEA in this cohort remain undefined.

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