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Multiple sclerosis and infants fed on diets deficient in vitamin A or in selenium and vitamin E

Medical Hypotheses
|May 1, 1982
PubMed

Insights

Early childhood nutrition, specifically insufficient vitamin A or selenium, may be a necessary factor for developing multiple sclerosis (MS) later in life. This hypothesis considers geographical and familial patterns of MS prevalence.

Area of Science:

  • Neurology
  • Nutritional Science
  • Epidemiology

Background:

  • Multiple sclerosis (MS) exhibits distinct epidemiological patterns, including geographical clustering and varying prevalence rates.
  • Familial tendencies and high prevalence in specific regions like the Orkney and Shetland Islands suggest complex etiological factors.
  • Existing research on MS and related conditions provides a basis for exploring nutritional influences.

Purpose of the Study:

  • To hypothesize the role of early childhood nutrition in the development of multiple sclerosis (MS).
  • To investigate the potential necessity of specific nutrient deficiencies in the onset of MS later in life.

Main Methods:

  • Analysis of characteristic features of MS, including geographical distribution and familial aggregation.
  • Consideration of existing research on MS and related subjects.
  • Formulation of a hypothesis based on observed epidemiological data.

Main Results:

  • MS prevalence decreases towards the equator among Caucasians.
  • Unusually high MS prevalence is noted in the Orkney and Shetland Islands.
  • A hypothesis is proposed linking early nutritional deficiencies to later MS onset.

Conclusions:

  • Insufficient intake of vitamin A or selenium during the first six months of infancy may be a necessary, though not sufficient, condition for developing MS.
  • Vitamin E's role in protecting vitamin A against degradation is also considered a potential contributing factor.

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