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[Kanamycin deposition in the peritoneum as affected by dimexide]

Antibiotiki
|February 1, 1981
PubMed

Insights

Dimethyl sulfoxide (DMSO) enhances kanamycin deposition in the peritoneum, contrary to expectations. This effect prolongs kanamycin retention time, particularly in cases of peritonitis.

Area of Science:

  • Pharmacology
  • Drug Absorption
  • Peritoneal Drug Delivery

Background:

  • Dimethyl sulfoxide (DMSO) is often considered to enhance drug absorption.
  • The peritoneal absorption kinetics of kanamycin require further investigation.

Purpose of the Study:

  • To investigate the effect of DMSO on kanamycin absorption from the peritoneum.
  • To evaluate the impact of peritonitis on this interaction.

Main Methods:

  • Intraperitoneal administration of DMSO and kanamycin in normal albino rats.
  • Induction of aseptic peritonitis in a subset of rats.
  • Measurement of kanamycin concentration in peritoneal fluid and blood over time.

Main Results:

  • DMSO administration led to significant kanamycin deposition within the peritoneum.
  • Kanamycin liberation into the bloodstream was slow and prolonged.
  • Peritoneal kanamycin concentrations increased 3-8 fold, extending retention by 10-13 hours.
  • The deposition effect was more pronounced in rats with peritonitis.

Conclusions:

  • DMSO inhibits rapid kanamycin absorption from the peritoneum, promoting local drug deposition.
  • This DMSO-induced peritoneal deposition of kanamycin offers a potential strategy for prolonged local drug therapy.
  • The findings challenge the general assumption of DMSO solely enhancing systemic drug absorption.

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