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Possible "atavistic" structures in human aneuploids

Insights

Aneuploid neonates (trisomy 18 and 13) exhibit unusual supernumerary muscles, some absent in humans but common in primates. These may represent atavistic structures, offering insights into human evolutionary development.

Area of Science:

  • Human Anatomy
  • Developmental Biology
  • Evolutionary Biology

Background:

  • Aneuploidy, such as trisomy 18 and 13, can lead to developmental anomalies.
  • Human anatomy occasionally presents variations, including supernumerary muscles.

Purpose of the Study:

  • To document and analyze supernumerary muscles found in neonates with trisomy 18 and 13.
  • To explore the potential evolutionary and developmental origins of these anatomical variations.

Main Methods:

  • Detailed anatomical dissection of aneuploid neonates.
  • Comparative analysis of identified muscles with primate anatomy.
  • Review of embryological and evolutionary mechanisms.

Main Results:

  • Identification of several supernumerary muscles (e.g., platysma occipitalis, rhomboidus occipitalis) not typically found in humans.
  • Observation of muscle absence (e.g., palmaris longus) in some specimens.
  • Presence of a linguofacial trunk in one specimen.
  • Noted similarity of these muscles to those regularly found in monkeys and apes.

Conclusions:

  • The supernumerary muscles in aneuploid neonates are suggested to be atavistic structures.
  • These findings provide insights into primate muscle homologies and developmental pathways.
  • Mechanisms for the development of these muscles in human aneuploids are proposed.

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