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Postmenopausal osteoporosis: prevention versus cure

L V Avioli

    Federation Proceedings
    |July 1, 1981
    PubMed
    Summary
    This summary is machine-generated.

    Postmenopausal women often suffer from vertebral and femoral neck fractures due to osteopenia. Ensuring adequate calcium intake from young adulthood is crucial for preventing bone mass loss and fractures.

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    Area of Science:

    • Gerontology
    • Orthopedics
    • Nutritional Science

    Background:

    • Osteopenia leads to vertebral and femoral neck fractures, significantly impacting postmenopausal women's health.
    • Bone mass loss begins in the fourth decade and accelerates in females, with multifactorial causes like lifestyle and genetics.
    • Current understanding of fracture-prone osteopenia pathogenesis remains incomplete.

    Purpose of the Study:

    • To highlight the critical role of calcium intake in preventing osteopenia and related fractures.
    • To emphasize the importance of nutritional adequacy, particularly calcium, for bone health in women across different life stages.

    Main Methods:

    • Review of existing literature on osteopenia, fracture syndromes, and risk factors.
    • Analysis of observations regarding calcium intake patterns in women.

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  • Synthesis of information on the impact of nutrition on bone metabolism.

    • Main Results:

    • Inadequate calcium intake in young women (20s-30s) and negative calcium balance during perimenopause are consistent findings.
    • Sedentary lifestyles, genetics, hormonal changes, vitamin deficiencies, diet, and smoking are implicated in osteopenia.
    • Calcium is identified as a key skeletal nutrient essential for bone health.

    Conclusions:

    • Adequate calcium nutrition is vital for all women, including young menstruating individuals and postmenopausal females.
    • Addressing calcium intake is a primary consideration for therapeutic strategies against osteopenia and fractures.
    • Further research is needed to establish a unified hypothesis for osteopenia pathogenesis.