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Related Experiment Videos

Congenital mirror movements

G D Schott, M A Wyke

    Journal of Neurology, Neurosurgery, and Psychiatry
    |July 1, 1981
    PubMed
    Summary
    This summary is machine-generated.

    This study examines congenital mirror movements, a type of abnormal synkinesia affecting hands. Findings suggest varied origins, including hereditary and sporadic cases, with potential links to impaired cerebral inhibition and abnormal motor pathways.

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    Area of Science:

    • Neurology
    • Neuroscience
    • Movement Disorders

    Background:

    • Congenital mirror movements are involuntary, symmetrical movements of one limb that mirror voluntary movements of the contralateral limb.
    • These movements represent a specific type of abnormal synkinesia, often affecting the hands.
    • The etiology can be hereditary, associated with underlying neurological abnormalities, or sporadic.

    Purpose of the Study:

    • To describe seven patients with congenital mirror movements.
    • To explore the heterogeneity and variability of this disorder.
    • To consider potential underlying mechanisms and neural pathways involved.

    Main Methods:

    • Clinical assessment of seven patients with congenital mirror movements.
    • Neuropsychological evaluation of affected individuals.

    Related Experiment Videos

  • Analysis of movement characteristics, including tone changes and modulation by neck position and biofeedback.

    • Main Results:

    • Seven cases of congenital mirror movements are presented, illustrating hereditary, neurological abnormality-associated, and sporadic forms.
    • Significant heterogeneity was observed, including increased muscle tone with arm movement and modulation of associated movements.
    • The study highlights the variability in presentation and potential for modification of mirror movements.

    Conclusions:

    • Congenital mirror movements can arise from diverse causes and exhibit marked variability.
    • Potential mechanisms include impaired cerebral inhibition and abnormal functioning of motor pathways.
    • The role of direct, crossed corticospinal pathways and preceding cerebral events is emphasized.