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Terbutaline and maternal cardiac function

J M Wagner, M J Morton, K A Johnson

    JAMA
    |December 11, 1981
    PubMed
    Summary
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    Terbutaline sulfate significantly increases heart rate and cardiac output in pregnant women with premature labor. These cardiac effects suggest it is a potent agent, likely not causing pulmonary edema through heart failure.

    Area of Science:

    • Cardiology
    • Obstetrics
    • Pharmacology

    Background:

    • Premature labor is a significant concern in obstetrics.
    • Terbutaline sulfate is commonly used to manage premature labor.
    • Understanding the cardiovascular effects of terbutaline during pregnancy is crucial for patient safety.

    Purpose of the Study:

    • To evaluate the impact of terbutaline sulfate on left ventricular size and performance.
    • To compare cardiovascular changes during terbutaline therapy versus oxytocin-induced uterine activity.
    • To determine if cardiac failure contributes to pulmonary edema associated with terbutaline.

    Main Methods:

    • M-mode echocardiography was utilized to assess cardiac parameters.
    • Pregnant women undergoing treatment for premature labor were studied.

    Related Experiment Videos

  • A comparison group received oxytocin for labor induction or challenge testing.

    • Main Results:

    • Terbutaline therapy led to significant increases in heart rate, ejection fraction, and cardiac output.
    • End-diastolic volume and systolic blood pressure remained unchanged.
    • Diastolic blood pressure and end-systolic volume decreased; no significant changes were observed with oxytocin.

    Conclusions:

    • Terbutaline sulfate acts as a potent inotropic and chronotropic agent in pregnant patients.
    • The observed cardiovascular changes do not indicate cardiac failure as a cause of pulmonary edema.
    • Terbutaline's cardiovascular profile requires careful consideration during premature labor management.