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The haemodynamic, histopathological and hormonal features of alcoholic cardiac beriberi

Insights

Cardiac beriberi, a form of heart failure in chronic alcoholics due to thiamine deficiency, can present with biventricular failure and elevated cardiac output. Diagnosis and treatment involve thiamine supplementation, with histological changes noted in myocardial biopsies.

Area of Science:

  • Cardiology
  • Nutritional Science
  • Internal Medicine

Background:

  • Cardiac beriberi is a potentially reversible cause of heart failure.
  • Chronic alcoholism is a significant risk factor for thiamine deficiency.
  • The condition may be underdiagnosed in clinical practice.

Purpose of the Study:

  • To describe clinical and pathological findings in five cases of cardiac beriberi.
  • To evaluate diagnostic methods, including hemodynamic studies and myocardial biopsy.
  • To assess the incidence and characteristics of cardiac beriberi in chronic alcoholics.

Main Methods:

  • Clinical case series of five chronic alcoholics with suspected cardiac beriberi.
  • Diagnostic workup included hemodynamic measurements, left ventriculography, coronary angiography, and endomyocardial biopsy.
  • Thiamine deficiency was assessed via dietary intake and response to thiamine therapy.
  • Transketolase assay and response to intravenous thiamine during catheterization were utilized.

Main Results:

  • Patients presented with biventricular failure, elevated cardiac output, and depressed left ventricular function.
  • Endomyocardial biopsies showed histological changes including vacuolation, edema, hypertrophy, fibrosis, and cellular infiltration.
  • No specific beriberi lesion was identified histologically, but biopsies were quantitatively abnormal.
  • Plasma renin, angiotensin II, and aldosterone levels were lower than in low-output heart failure.

Conclusions:

  • Cardiac beriberi is a significant cause of heart failure in chronic alcoholics, potentially more common than realized.
  • Clinical diagnosis relies on biventricular failure, low thiamine intake, and response to thiamine.
  • Hemodynamic studies, transketolase testing, and myocardial biopsy aid in diagnosis and understanding pathophysiology.

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