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T-cell development of resistance to apoptosis is driven by a metabolic shift in carbon source and altered activation of death pathways.

Cell death and differentiation·2015

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Cell death and differentiation·2014

Selective glucocorticoid receptor translational isoforms reveal glucocorticoid-induced apoptotic transcriptomes.

Cell death & disease·2013

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Minerva endocrinologica·2010

F-actin-dependent activation of NADPH oxidase 5 by the vacuolar-type ATPase.

The Journal of biological chemistry·2026

Enhanced SIRT3 activity potentiates germinal center epigenetic modulation through α-ketoglutarate.

The Journal of biological chemistry·2026

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The Journal of biological chemistry·2026

Genetic and biochemical screens identify MGAT1 as a druggable glycosyltransferase target in STK11-mutant lung cancer.

The Journal of biological chemistry·2026

GATA3-driven CDC20 activation facilitates tumor growth and metastasis through suppressing Bax/Bcl-2 ratio and boosting EMT process in neuroblastoma.

The Journal of biological chemistry·2026

5-Iodotubercidin inhibits Epithelial to Mesenchymal Transition by inhibiting IKK/NFκB-dependent gene expression.

The Journal of biological chemistry·2026

E. coli mutant NR71 grows on succinate in the absence of an F1 ATPase.

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Journal of materials chemistry. B·2026

B-active-center-driven photocatalytic coupling of CO2 and N2 toward urea synthesis on penta-B2C.

Nanoscale·2026

Key enzyme optimization and multi-node metabolic flux regulation drive l-arginine production in Escherichia coli.

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Applied radiation and isotopes : including data, instrumentation and methods for use in agriculture, industry and medicine·2026

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Specific modification of rat uterine estrogen receptor by pyridoxal 5'-phosphate

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microRNA-17-5p modulates ventral hippocampal transcriptome and synaptic proteome: Implications for emotional regulation in adult male rats.

MiR-16 mediates trastuzumab and lapatinib response in ErbB-2-positive breast and gastric cancer via its novel targets CCNJ and FUBP1.

T-cell development of resistance to apoptosis is driven by a metabolic shift in carbon source and altered activation of death pathways.

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Specific modification of rat uterine estrogen receptor by pyridoxal 5'-phosphate

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