Related Experiment Videos

Complementation analysis of measles virus mutants isolated from persistently infected lymphoblastoid cell lines

Journal of Virology
|March 1, 1980
PubMed

Insights

Measles virus in human cells produces varied, temperature-sensitive particles. These measles virus mutants show genetic diversity and can interfere with normal virus production, impacting infection dynamics.

Area of Science:

  • Virology
  • Cell Biology
  • Immunology

Background:

  • Human lymphoblastoid cell lines can harbor persistent measles virus infections.
  • Measles virus (MeV) virions released from infected cells are often heterogeneous.

Purpose of the Study:

  • To characterize the temperature-sensitive (ts) measles virus mutants released from persistently infected human lymphoblastoid cell lines.
  • To investigate the genetic diversity and functional properties of these MeV mutants.

Main Methods:

  • Isolation and plaque purification of temperature-sensitive measles virus mutants.
  • Efficiency of plating assays at permissive (31°C) and non-permissive (39°C) temperatures.
  • Complementation analysis to determine genetic relatedness of mutants.
  • Assessment of antigen and RNA production at elevated temperatures.

Main Results:

  • Over 80% of infectious MeV particles were temperature-sensitive for plaque formation at 39°C.
  • Isolated ts MeV mutants exhibited heterogeneity in plating efficiency, antigen, and RNA production.
  • Complementation analysis revealed at least four previously described complementation groups, with potential new groups identified.
  • A majority of mutants possessed multiple mutations, with RNA-deficient (Group B) mutants being predominant.
  • Two ts isolates demonstrated interference with parental MeV production.

Conclusions:

  • Persistently infected human lymphoblastoid cells release genetically diverse and functionally heterogeneous temperature-sensitive measles virus populations.
  • The identified measles virus mutants, particularly RNA-deficient ones, may play a role in viral persistence and interference phenomena.
  • Further characterization of these novel or Group E mutants could elucidate mechanisms of measles virus persistence and pathogenesis.

Related Concept Videos