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Enkephalin mydriasis in mice

Insights

Enkephalins, similar to morphine, cause mydriasis (pupil dilation) in mice. However, enkephalins are not involved in the natural regulation of pupillary diameter.

Area of Science:

  • Pharmacology
  • Neuroscience
  • Ophthalmology

Background:

  • Morphine is a known mydriatic agent in mice.
  • Enkephalins are endogenous opioid peptides with various physiological roles.

Purpose of the Study:

  • To compare the mydriatic potency and time course of enkephalins versus morphine.
  • To investigate the potential physiological role of enkephalins in pupillary diameter regulation.

Main Methods:

  • Administered morphine, D-Ala-D-Leu-enkephalin, Met-enkephalin, and Leu-enkephalin to mice to assess mydriasis.
  • Compared the potency and duration of mydriatic effects.
  • Administered naloxone or D-phenylalanine to mice to evaluate the involvement of enkephalins in physiological pupillary control.

Main Results:

  • Enkephalins induced mydriasis in mice, with varying potencies relative to morphine.
  • The duration of mydriasis was shortest for enkephalins and longest for morphine.
  • Naloxone antagonized the mydriatic effects of all tested agents similarly.
  • Administration of naloxone or D-phenylalanine did not alter basal pupillary diameter in mice.

Conclusions:

  • Enkephalins possess mydriatic properties in mice.
  • The findings suggest that enkephalins are unlikely to be involved in the physiological regulation of pupillary diameter.

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