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Size at birth and plasma insulin-like growth factor-1 concentrations

C H Fall1, A N Pandit, C M Law

  • 1MRC Environmental Epidemiology Unit, Southampton General Hospital.

Insights

Children born with lower birth weight have higher insulin-like growth factor-1 (IGF-1) levels in childhood. This suggests fetal under-nutrition may alter the IGF-1 axis, potentially linking to later health issues like high blood pressure.

Area of Science:

  • Pediatric Endocrinology
  • Developmental Biology
  • Human Growth and Development

Background:

  • Reduced fetal growth is a concern for long-term health.
  • Insulin-like growth factor-1 (IGF-1) plays a crucial role in growth and development.
  • The relationship between fetal growth and childhood IGF-1 levels requires further investigation.

Purpose of the Study:

  • To investigate the hypothesis that reduced fetal growth results in altered plasma IGF-1 concentrations during childhood.
  • To explore the association between birth weight and childhood IGF-1 levels, adjusting for current body size and sex.

Main Methods:

  • A follow-up study involving children from Pune, India, and Salisbury, England.
  • Measurements included birth weight, current weight and height, and plasma IGF-1 concentrations.
  • Statistical analysis was performed to determine the relationship between birth weight and IGF-1, controlling for covariates.

Main Results:

  • Plasma IGF-1 concentrations were positively correlated with current height and weight, and higher in girls than boys.
  • After adjusting for sex and current size, IGF-1 concentrations were inversely related to birth weight in both study groups.
  • Children with lower birth weight exhibited higher IGF-1 concentrations relative to their current size.

Conclusions:

  • Lower birth weight is associated with elevated childhood IGF-1 concentrations, supporting the hypothesis of in utero reprogramming of the IGF-1 axis.
  • Increased IGF-1 may be linked to postnatal catch-up growth and could be a mechanism connecting reduced fetal growth to hypertension later in life.
Abstract

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