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[Amylasemia in uremics: a complex interpretation]

M Buemi1, M Molinaro, R Timpanelli

  • 1Dipartimento di Medicina Interna, Università di Messina.

Rivista Europea Per Le Scienze Mediche E Farmacologiche = European Review for Medical and Pharmacological Sciences = Revue Europeenne Pour Les Sciences Medicales Et Pharmacologiques
|January 1, 1994
PubMed
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Uremia often elevates plasmatic amylase, potentially linked to hyperparathyroidism. However, calcium antagonists did not alter enzyme levels, suggesting hyperparathyroidism influences pancreatic secretion but not enzyme levels in uremic patients.

Area of Science:

  • Nephrology
  • Endocrinology
  • Biochemistry

Background:

  • Elevated plasmatic amylase is common in uremia.
  • Hyperparathyroidism is frequently observed in uremic patients and implicated in amylase elevation via increased cytosolic calcium.
  • The precise relationship between hyperparathyroidism, calcium metabolism, and pancreatic enzyme levels in uremia requires further elucidation.

Purpose of the Study:

  • To investigate the correlation between plasmatic amylase, lipase, calcium, phosphates, and parathormone levels in uremic patients.
  • To assess the effect of calcium antagonists (Verapamil and Diltiazem) on these parameters.

Main Methods:

  • Measurements of plasmatic amylase, lipase, calcium, phosphates, and parathormone were performed before and after administration of calcium antagonists.

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  • The study population comprised uremic patients with hyperparathyroidism.

    • Main Results:

    • No significant correlation was found between the measured parameters and calcium antagonist administration.
    • Hyperparathyroidism was observed to increase pancreatic secretion.
    • Hyperparathyroidism did not appear to influence the overall amount of pancreatic enzymes in uremic patients.

    Conclusions:

    • Calcium antagonists do not seem to modulate plasmatic amylase or lipase levels in uremic patients with hyperparathyroidism.
    • While hyperparathyroidism affects pancreatic secretion, its direct impact on the total enzyme levels in uremia is limited.
    • Further research is needed to fully understand the complex interplay of factors influencing enzyme levels in uremic conditions.