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Quantification of group A colicin import sites

D Duché1, L Letellier, V Géli

  • 1Laboratorie d'Ingénierie et de Dynamique des Systèmes Membranaires, Centre National de la Recherche Scientifique, UPR 9027, Marseille, France.

Journal of Bacteriology
|September 1, 1995
PubMed

Insights

A pore-forming colicin A mutant, unable to form channels, still binds receptors. This finding helps estimate the number of colicin A receptors and translocation sites on Escherichia coli cells.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Biophysics

Background:

  • Pore-forming colicins are bacteriocins that create ion channels in the Escherichia coli inner membrane.
  • Colicin translocation involves outer membrane receptors and translocation machinery, with two groups (A and B) utilizing different protein pathways (Tol or TonB/ExbB/ExbD).

Purpose of the Study:

  • To investigate the role of the pore-forming domain in colicin translocation and receptor interaction.
  • To quantify the number of colicin A receptors and translocation sites on Escherichia coli.

Main Methods:

  • Utilized a double-cysteine colicin A mutant with a disulfide bond in its pore-forming domain.
  • Measured colicin-induced K+ efflux and performed competition experiments with wild-type colicins and the mutant.

Main Results:

  • The colicin A mutant was translocated through the envelope but could not form an inner membrane channel.
  • Preincubation with the mutant blocked group A colicin binding but not group B, indicating specific interactions.
  • The mutant remained associated with the receptor and import machinery during inner membrane interaction.

Conclusions:

  • Escherichia coli cells possess approximately 400 colicin A receptors.
  • Each Escherichia coli cell has approximately 1,000 colicin A translocation sites.

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