Related Experiment Video

Updated: Sep 10, 2026

Fecal Glucocorticoid Analysis: Non-invasive Adrenal Monitoring in Equids
08:02

Fecal Glucocorticoid Analysis: Non-invasive Adrenal Monitoring in Equids

Published on: April 25, 2016

Adrenal hyperandrogenism: detection by adrenal scintigraphy

J E Freitas, W H Beierwaltes, R H Nishiyama

    Journal of Endocrinological Investigation
    |January 1, 1978
    PubMed

    More Related Videos

    A Novel Method: Super-selective Adrenal Venous Sampling06:08

    A Novel Method: Super-selective Adrenal Venous Sampling

    Published on: September 15, 2017

    Isolation, Fixation, and Immunofluorescence Imaging of Mouse Adrenal Glands08:37

    Isolation, Fixation, and Immunofluorescence Imaging of Mouse Adrenal Glands

    Published on: October 2, 2018

    Related Concept Videos

    Imaging Studies II: Positron Emission Tomography and Scintigraphy01:25

    Imaging Studies II: Positron Emission Tomography and Scintigraphy

    Positron Emission Tomography (PET) is a medical imaging technique that provides crucial insights into the body's physiological functions at a molecular level. It is an indispensable resource for diagnosing, staging, and monitoring various illnesses, notably cancer, neurological disorders, and cardiovascular conditions.
    Fundamental Principles of PET
    Cushing Syndrome I: Introduction01:26

    Cushing Syndrome I: Introduction

    Cushing syndrome refers to the collection of clinical manifestations that arise when tissues are exposed to excessive amounts of cortisol or cortisol-like medications over an extended period. Cortisol, a glucocorticoid produced by the adrenal cortex, regulates metabolism, immune responses, and the body’s adaptation to stress. When its concentration remains chronically elevated, these physiological pathways become dysregulated, resulting in the characteristic features of the syndrome.Exogenous...
    Cushing Syndrome II: Pathophysiology01:19

    Cushing Syndrome II: Pathophysiology

    Cortisol production is normally governed by the hypothalamic–pituitary–adrenal (HPA) axis, which maintains hormonal balance through tightly regulated feedback mechanisms. Disruption of this regulatory system is central to the development of Cushing syndrome, whether the excess cortisol originates from external medications or internal pathology. Persistent cortisol elevation alters metabolism, immune function, and endocrine signaling, producing the characteristic clinical features of the...