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Identification of the epitope recognized by the human V beta 5-specific monoclonal antibody 42/1C1. Potential

J Henwood1, J C Goodall, A W Boylston

  • 1Department of Rheumatology, University of Birmingham, England.

Human Immunology
|April 1, 1995
PubMed

Insights

Researchers identified a new T-cell receptor (TCR) variant recognizing mycobacterial stress protein. This discovery highlights challenges for monoclonal antibody therapies in treating diseases by differentiating similar TCRs.

Area of Science:

  • Immunology
  • Molecular Biology
  • Microbiology

Background:

  • T-cell receptors (TCRs) are crucial for adaptive immunity, recognizing specific peptide-MHC complexes.
  • Mycobacterial 65-kd heat shock proteins are known antigens involved in immune responses.
  • The V beta 5 family of TCRs plays a role in recognizing mycobacterial antigens.

Purpose of the Study:

  • To characterize T-cell clones specific for the mycobacterial 65-kd stress protein.
  • To investigate the diversity within the V beta 5 family of TCRs.
  • To identify the epitope recognized by the monoclonal antibody (mAb) 42/1C1.

Main Methods:

  • Generation of T-cell clones specific for residues 4-13 of the mycobacterial 65-kd stress protein.
  • Polymerase Chain Reaction (PCR) to define V beta 5 family expression.
  • Flow cytometry using mAb 42/1C1 for T-cell clone staining.
  • TCR sequencing to identify novel V beta 5 family members.

Main Results:

  • All generated T-cell clones expressed a V beta 5 family member.
  • A subset of clones, not recognized by mAb 42/1C1, expressed a previously unidentified V beta 5 family member.
  • Comparison of TCR sequences allowed identification of a probable epitope for mAb 42/1C1.
  • Identical MHC-peptide recognition by T cells can involve highly similar, yet distinguishable, TCRs.

Conclusions:

  • A novel V beta 5 family member and its corresponding TCRs were identified.
  • The study identified a probable epitope for mAb 42/1C1, differentiating TCRs within the V beta 5 family.
  • Subtle differences in TCRs recognizing the same antigen may complicate the use of monoclonal antibodies in disease therapy.

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