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Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor

A J Hörlein1, A M Näär, T Heinzel

  • 1Howard Hughes Medical Institute, University of California, San Diego, La Jolla 92093-0648, USA.

Nature
|October 5, 1995
PubMed

Insights

A newly identified nuclear receptor co-repressor (N-CoR) inhibits gene transcription by thyroid-hormone and retinoic-acid receptors. This finding suggests analogous repression mechanisms across species for these crucial hormone receptors.

Area of Science:

  • Molecular Biology
  • Genetics
  • Endocrinology

Background:

  • Thyroid-hormone and retinoic-acid receptors regulate gene expression as activators and repressors.
  • Understanding the mechanisms of gene repression is critical for cellular regulation.

Purpose of the Study:

  • To identify and characterize co-repressors involved in nuclear receptor-mediated gene silencing.
  • To elucidate the molecular mechanisms underlying ligand-independent repression by thyroid-hormone and retinoic-acid receptors.

Main Methods:

  • Biochemical isolation and characterization of a novel nuclear receptor co-repressor.
  • Assays to determine the role of the identified co-repressor in gene transcription inhibition.
  • Comparative analysis of repression mechanisms across different organisms.

Main Results:

  • Identification of a 270K nuclear receptor co-repressor (N-CoR).
  • Demonstration that N-CoR mediates ligand-independent inhibition of gene transcription.
  • Evidence suggesting conserved co-repressor-dependent repression mechanisms between mammalian receptors and those in yeast and Drosophila.

Conclusions:

  • A novel co-repressor, N-CoR, plays a significant role in regulating gene expression by thyroid-hormone and retinoic-acid receptors.
  • The mechanisms of repression by these nuclear receptors are conserved across evolution.
  • This discovery provides insights into the fundamental processes of gene regulation and potential therapeutic targets.

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