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Plasma interleukin 8 and polymorphonuclear leukocyte elastase concentrations in patients with septic shock

S Endo1, K Inada, M Ceska

  • 1Critical Care and Emergency Center, Iwate Medical University, Morioka, Japan.

Journal of Inflammation
|January 1, 1995
PubMed

Insights

Elevated levels of interleukin 8 (IL-8) and polymorphonuclear leukocyte elastase (PMNE) are linked to septic shock and adult respiratory distress syndrome (ARDS). These inflammatory markers correlate with disease severity and endotoxin levels.

Area of Science:

  • Biochemistry
  • Immunology
  • Critical Care Medicine

Background:

  • Septic shock is a life-threatening condition characterized by circulatory dysfunction.
  • Adult respiratory distress syndrome (ARDS) is a common and severe complication of septic shock.
  • The roles of specific inflammatory mediators in septic shock and ARDS require further elucidation.

Purpose of the Study:

  • To investigate the plasma concentrations of interleukin 8 (IL-8) and polymorphonuclear leukocyte elastase (PMNE) in patients with septic shock.
  • To determine the correlation between IL-8, PMNE, and endotoxin levels in septic shock.
  • To explore the relationship between PMNE and the oxygenation index in septic shock.

Main Methods:

  • Plasma concentrations of IL-8, PMNE, and endotoxin were measured in patients with and without septic shock.
  • Statistical analyses, including correlation coefficients and significance testing, were employed.
  • The oxygenation index (PaO2/FiO2) was assessed at the onset of septic shock.

Main Results:

  • IL-8 concentrations were significantly higher in patients with septic shock compared to those without shock (P < 0.0001).
  • Significant positive correlations were observed between IL-8 and PMNE (r = 0.6916, P < 0.0001) and between IL-8 and endotoxin (r = 0.5584, P = 0.0016).
  • A significant negative correlation was found between serum PMNE and the oxygenation index (r = -0.8237, P < 0.0001).

Conclusions:

  • IL-8 and PMNE are produced in substantial amounts during septic shock.
  • These inflammatory mediators may contribute to the pathogenesis of septic shock and ARDS.
  • The findings highlight the potential involvement of IL-8 and PMNE in the development of septic ARDS.

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