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Changes of circadian blood pressure patterns and cardiovascular parameters indicate lateralization of sympathetic

D Sander1, J Klingelhöfer

  • 1Department of Neurology, Technical University of Munich, Germany.

Insights

Right-sided strokes significantly impact sympathetic nervous system activity, leading to reduced blood pressure variability and increased cardiac risks. This suggests a dominant role of the right hemisphere in sympathetic responses post-stroke.

Area of Science:

  • Neurology
  • Cardiology
  • Autonomic Nervous System

Background:

  • Investigating hemispheric asymmetry in stroke consequences on cardiovascular regulation.
  • Examining sympathetic nervous system responses following left- versus right-sided brain infarction.

Observation:

  • 35 patients with hemispheric brain infarction were analyzed for circadian blood pressure and cardiovascular variability.
  • No significant differences in age, infarction size, or insular cortex damage were found between groups.
  • Right-sided infarction correlated with reduced diastolic blood pressure variability and increased nocturnal blood pressure elevation.

Findings:

  • Patients with right-sided infarction exhibited significantly lower circadian blood pressure variability and a higher incidence of nocturnal blood pressure increase.
  • Elevated serum noradrenaline levels, QT prolongation, and cardiac arrhythmias were more frequent in patients with right-sided infarction.
  • While insular infarction caused pronounced changes regardless of hemisphere, right-sided strokes involving the insula led to myocardial infarction in 13% of cases.

Implications:

  • Findings suggest a lateralization of sympathetic activation, with right-sided brain infarction demonstrating a dominant sympathetic effect.
  • This lateralization has potential implications for understanding stroke-related cardiovascular complications and developing targeted therapies.
  • Further research is warranted to explore the mechanisms behind right-hemispheric dominance in sympathetic responses to stroke.

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