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Polymorphonuclear leucocyte dysregulation in patients with gram-negative septicaemia assessed by flow cytometry

C Wenisch1, P Parschalk, M Hasenhündl

  • 1Department of Infectious Diseases, University Hospital of Vienna, Austria.

Insights

Granulocytes in patients with gram-negative septicaemia show impaired phagocytic function and reactive oxygen intermediate (ROI) release. Therapy improved these functions, suggesting granulocytes are both participants and targets in the septic inflammatory response.

Area of Science:

  • Immunology
  • Hematology
  • Infectious Diseases

Background:

  • Gram-negative septicaemia is a severe infection characterized by a systemic inflammatory response.
  • Granulocytes play a crucial role in combating bacterial infections through phagocytosis and the production of reactive oxygen intermediates (ROI).
  • The functional status of granulocytes during septicaemia and the impact of therapy are not fully understood.

Purpose of the Study:

  • To investigate the phagocytic capacity and ROI production of granulocytes in patients with gram-negative septicaemia.
  • To compare granulocyte function in septic patients with healthy controls.
  • To evaluate the effect of therapy on granulocyte function during gram-negative septicaemia.

Main Methods:

  • Flow cytometry was utilized to assess granulocyte function.
  • Phagocytic capacity was measured by the uptake of fluorescein isothiocyanate (FITC)-labelled Escherichia coli.
  • ROI generation was quantified by measuring the conversion of dihydrorhodamine 123 to rhodamine 123.

Main Results:

  • Granulocytes from patients with gram-negative septicaemia demonstrated reduced phagocytosis of E. coli and diminished ROI production compared to healthy controls.
  • Initiation of therapy led to significant increases in both phagocytosis and ROI production.
  • Granulocyte function normalized within 7 days of treatment.

Conclusions:

  • Granulocytes exhibit impaired function during gram-negative septicaemia, indicating they are affected by the septic inflammatory response.
  • Therapeutic interventions can restore granulocyte phagocytic capacity and ROI generation.
  • These findings highlight the complex role of granulocytes in sepsis, acting as both effector cells and targets of the host response.

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