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Staphylococcus aureus triggered reactive arthritis

A R Siam¹, M Hammoudeh

¹Department of Internal Medicine, Hamad Medical Corporation, Doha, Qatar.

Annals of the Rheumatic Diseases

|February 1, 1995

Summary

This summary is machine-generated.

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Reactive arthritis can rarely develop after Staphylococcus aureus infections. In these cases, being HLA-B27 negative may indicate a self-limited joint condition.

Area of Science:

Rheumatology
Infectious Diseases

Background:

Reactive arthritis is a form of inflammatory arthritis that affects joints.
It is triggered by an infection elsewhere in the body, often in the gastrointestinal or genitourinary tract.

Observation:

This report details two adult female patients who developed sterile arthritis following Staphylococcus aureus (Staph aureus) infections.
The first patient experienced two episodes of arthritis, linked to olecranon bursitis and a central venous catheter infection.
The second patient developed arthritis during treatment for pyomyositis.

Findings:

Both patients presented with self-limited arthritis and were negative for the HLA-B27 gene.
These cases suggest a rare association between Staph aureus infection and the development of reactive arthritis.

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Implications:

Reactive arthritis following Staph aureus infection is uncommon but possible.
HLA-B27 negativity may correlate with a milder, self-limiting course of reactive arthritis in these specific cases.