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Induction of apoptosis by the Bcl-2 homologue Bak

T Chittenden1, E A Harrington, R O'Connor

  • 1Apoptosis Technology Inc., Cambridge, Massachusetts 02139, USA.

Nature
|April 20, 1995
PubMed

Insights

Scientists discovered Bak, a new protein that promotes apoptosis (programmed cell death). Bak counteracts Bcl-2

Area of Science:

  • Molecular Biology
  • Cell Biology
  • Oncology

Background:

  • Apoptosis, or programmed cell death, is a critical cellular process.
  • Suppression of apoptosis is linked to cancer development.
  • The Bcl-2 oncogene inhibits apoptosis and is implicated in lymphomas.

Purpose of the Study:

  • To clone and functionally analyze a novel Bcl-2 homologue, named Bak.
  • To investigate Bak's role in apoptosis and its interaction with Bcl-2.

Main Methods:

  • Complementary DNA (cDNA) cloning of the Bak gene.
  • Functional analysis of Bak's role in promoting cell death.
  • Enforced expression of Bak in fibroblasts.

Main Results:

  • Bak was cloned and identified as a Bcl-2 homologue.
  • Bak actively promotes apoptosis, counteracting Bcl-2's anti-apoptotic effects.
  • Forced Bak expression led to rapid apoptosis in serum-deprived fibroblasts.

Conclusions:

  • Bak is a novel protein that promotes programmed cell death.
  • Bak may play a direct role in activating the cell death machinery.
  • Bak represents a potential target for cancer therapies by inducing apoptosis.

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