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Related Experiment Videos

Contralateral hyperhidrosis after cerebral infarction. Clinicoanatomic correlations in five cases

B S Kim1, Y I Kim, K S Lee

  • 1Department of Neurology, Catholic University Medical College, Seoul, Korea.

Stroke
|May 1, 1995
PubMed
Summary

Stroke can cause excessive sweating, particularly on one side of the body. This study identifies a potential pathway in the brainstem controlling sweating, offering new insights into stroke-related autonomic dysfunction.

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Area of Science:

  • Neurology
  • Autonomic Nervous System Research
  • Stroke Medicine

Background:

  • Excessive sweating (hyperhidrosis) post-stroke is rare and poorly understood.
  • Medullary infarction causing isolated contralateral hemihyperhidrosis without Horner's syndrome is unreported.
  • This study aims to improve recognition of stroke-induced hyperhidrosis.

Observation:

  • Five patients with stroke-induced hyperhidrosis were studied.
  • Contralateral hyperhidrosis occurred in patients with middle cerebral artery territory strokes and medullary infarctions.
  • One patient with basilar artery thrombosis experienced bilateral facial hyperhidrosis.

Findings:

  • Hyperhidrosis was transient, affecting the face and arm.
  • No associated autonomic dysfunction like Horner's syndrome or hypothalamic issues was observed.

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  • Medullary infarction can cause isolated contralateral hemihyperhidrosis.

    • Implications:

    • Suggests a lesion in a putative sympathoinhibitory pathway controlling sweating.
    • This pathway may descend from the cortex (possibly operculum) to the contralateral thoracic spinal cord.
    • Pathway fibers may run close to the corticospinal tract, explaining contralateral deficits.