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[The factors in decreased blood flow in the carotid arteries]
Insights
Reduced carotid artery (CA) circulation volume (CV) is linked to acute cerebral circulation disturbance (ACCD) and vascular dementia (VD). Arterial hypertension, not CA stenosis or cardiac issues, appears to be a key factor in lowering CA CV.
Area of Science:
- Neurology
- Vascular Medicine
- Medical Imaging
Background:
- Acute cerebral circulation disturbance (ACCD) and vascular dementia (VD) significantly impact patient health.
- Understanding the hemodynamics in carotid arteries (CA) is crucial for diagnosing and managing cerebrovascular diseases.
Purpose of the Study:
- To investigate the circulation volume (CV) in the carotid arteries (CA) of patients with ACCD and VD.
- To identify factors associated with reduced CV in these patient groups.
Main Methods:
- Ultrasonic duplex scanning was employed to measure CV in the CA.
- Echocardiography was used to assess cardiac function in patients.
- The study included 219 patients with ACCD, 26 with VD, and 54 healthy controls.
Main Results:
- Reduced CV in CA was observed in both ACCD and VD patients, particularly those with CA stenosis.
- For VD patients, a more severe drop in CA CV was not directly correlated with increasing CA occlusion.
- In patients without significant cardiac conditions or CA stenosis, arterial hypertension was prevalent and associated with lower CA CV.
Conclusions:
- Reduced CA CV is a notable finding in patients with ACCD and VD.
- Arterial hypertension emerges as a significant factor contributing to decreased CA CV, independent of CA stenosis or cardiac dysfunction.
- Further research into the role of hypertension in cerebrovascular hemodynamics is warranted.
Abstract:
The circulation volume (CV) in the carotid arteries (CA) has been studied at ultrasonic duplex scanning in 219 patients with a history of acute cerebral circulation disturbance (ACCD), 26 patients with vascular dementia (VD) and in 54 healthy controls. Reduced CV in CA was registered both in ACCD and VD patients who had or were of CA stenosis. More severe fall of CV in CA for VD patients was not associated with increasing occlusion of CA. In the absence of obvious cardiac affection (fibrillation, myocardial infarction) in ACCD and VD patients echocardiography recorded insignificant reduction of myocardial contractility and cardiac output. The majority of these patients without cardiac disease or CA stenosis had arterial hypertension which is supposed to be an essential factor of CV lowering in CA.