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Zinc needs and homeostasis during lactation

P B Moser-Veillon1

  • 1Department of Nutrition and Food Science, University of Maryland, College Park, 20742, USA.

The Analyst
|March 1, 1995
PubMed
Summary

Lactating women lose essential zinc into milk. Homeostatic mechanisms like increased absorption, reduced excretion, and bone resorption help compensate for this zinc loss, influencing dietary needs.

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Area of Science:

  • Human Physiology
  • Nutritional Science
  • Trace Element Metabolism

Background:

  • Lactation significantly increases maternal zinc loss through milk secretion.
  • Maternal zinc requirements rise during lactation, necessitating dietary adjustments or compensatory physiological mechanisms.

Purpose of the Study:

  • To investigate the homeostatic mechanisms that compensate for increased maternal zinc loss during lactation.
  • To understand how enhanced zinc absorption, reduced excretion, and bone resorption contribute to maintaining zinc balance.

Main Methods:

  • Review of existing literature on maternal zinc metabolism during lactation.
  • Analysis of studies reporting zinc absorption, urinary excretion, and bone mineral changes in lactating women.

Main Results:

  • Lactating women exhibit enhanced zinc absorption, particularly when dietary intake is low.
  • Urinary zinc excretion is significantly reduced postpartum.
  • Maternal bone resorption contributes zinc, which may be incorporated into milk.

Conclusions:

  • Homeostatic mechanisms, including enhanced absorption, reduced urinary excretion, and bone resorption, partially compensate for zinc secretion into milk.
  • These compensatory mechanisms are crucial for determining appropriate dietary recommendations for zinc intake in lactating women.

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