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[Sneddon syndrome: 9 cases]

J C Antoine1, D Michel, P Garnier

  • 1Service de Neurologie, Hôpital de Bellevue, Saint-Etienne.

Revue Neurologique
|June 1, 1994
PubMed
Summary
This summary is machine-generated.

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Sneddon's syndrome involves cerebrovascular events and livedo racemosa. This study highlights its complex pathophysiology, with varied neurological and autoimmune manifestations in nine patients.

Area of Science:

  • Neurology
  • Rheumatology
  • Vascular Medicine

Context:

  • Sneddon's syndrome is a rare condition characterized by livedo racemosa and recurrent cerebrovascular events.
  • The underlying pathophysiology remains incompletely understood, often involving autoimmune and thrombotic factors.

Purpose:

  • To describe the clinical, radiological, and immunological findings in a series of patients with Sneddon's syndrome.
  • To elucidate the heterogeneous presentation and complex pathophysiology of this syndrome.

Summary:

  • Nine patients (1 male, 8 females, mean age 49) with Sneddon's syndrome were analyzed.
  • Manifestations included cerebrovascular events (stroke, vascular dementia), livedo racemosa, and associated conditions like anticardiolipin antibodies, lupus erythematosus, and mitral valve defects.

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  • Cerebral angiography revealed arterial occlusions or moya-moya patterns.

    • Impact:

    • This study underscores the diverse clinical spectrum of Sneddon's syndrome.
    • It emphasizes the need for comprehensive evaluation, including neurological, rheumatological, and cardiovascular assessments, to manage this complex condition effectively.