Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Neurophysiologic studies of temporomandibular joint dysfunction

R Yemm

    Oral Sciences Reviews
    |January 1, 1976
    PubMed
    Summary

    Temporomandibular joint dysfunction may not stem from condyle displacement or malocclusion. Instead, central nervous system hyperactivity in jaw muscles could cause damage, leading to pain and dysfunction.

    Related Concept Videos

    You might also read

    Related Articles

    Articles linked to this work by shared authors, journal, and citation graph.

    Sort by
    Same author

    The use of silicone dentures for edentulous patients.

    Dental update·1998
    Same author

    Insulin-like growth factor (IGF)-I and -II and IGFBP secretion by ovine satellite cell strains grown alone or in coculture with 3T3-L1 preadipocytes.

    In vitro cellular & developmental biology. Animal·1998
    Same author

    Electromyographic evidence for a digastric reflex evoked by perioral stimuli in humans.

    Journal of oral rehabilitation·1997
    Same author

    Twitch interpolation in the assessment of the maximum force-generating capacity of the jaw-closing muscles in man.

    Archives of oral biology·1996
    Same author

    Removable partial denture production in Scotland.

    Quintessence international (Berlin, Germany : 1985)·1996
    Same author

    Changes in human jaw muscles with age and dental state.

    Gerodontology·1993

    Area of Science:

    • Physiology
    • Neurology
    • Dentistry

    Background:

    • Temporomandibular joint (TMJ) dysfunction is a common clinical issue.
    • Existing theories often link TMJ dysfunction to condylar displacement or dental malocclusion.
    • The physiological underpinnings require further elucidation.

    Purpose of the Study:

    • To critically evaluate the experimental evidence for proposed physiological causes of TMJ dysfunction.
    • To investigate the role of muscle hyperactivity and its origins in TMJ dysfunction.
    • To correlate central nervous system activity with TMJ dysfunction.

    Main Methods:

    • Review and synthesis of existing experimental and clinical evidence.
    • Analysis of physiological mechanisms related to jaw muscle activity.
    • Evaluation of the link between malocclusion, reflex mechanisms, and muscle hyperactivity.

    Main Results:

    • Little evidence supports condylar displacement as a primary cause of TMJ dysfunction.
    • Experimental data offers some support for muscle hyperactivity, but not clearly linked to malocclusion via reflexes.
    • Growing evidence suggests central nervous system origin for jaw muscle hyperactivity.

    Conclusions:

    • Centrally induced hyperactivity of jaw closing muscles may be sufficient to cause muscle damage.
    • This muscle damage can lead to impaired function, local pain, and referred pain.
    • Revising the understanding of TMJ dysfunction etiology is warranted.

    Related Experiment Videos