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Stimulation of brush border enzyme activity along the rat small intestine by misoprostol

A Martí1, M P Fernández-Otero

  • 1Departamento de Fisiología y Nutrición, Universidad de Navarra, Pamplona, Spain.

Revista Espanola De Fisiologia
|June 1, 1994
PubMed

Insights

Misoprostol significantly enhanced digestive enzyme activity in growing rats, increasing brush border enzymes like sucrase and maltase. This suggests improved intestinal digestive function following treatment.

Area of Science:

  • Gastroenterology
  • Pharmacology
  • Biochemistry

Background:

  • Brush border membrane enzymes are crucial for nutrient digestion and absorption.
  • Misoprostol is a prostaglandin E1 analog with known gastroprotective effects.

Purpose of the Study:

  • To investigate the impact of Misoprostol on brush border enzyme activity in growing rats.
  • To determine if Misoprostol enhances the digestive capacity of the small intestine.

Main Methods:

  • Growing rats were orally administered Misoprostol (0.3 mg/kg b.w.) for four weeks.
  • Relative organ weights and mucosal weights of the stomach and small intestine were measured.
  • Specific activities of disaccharidases (sucrase, maltase), alkaline phosphatase, and aminopeptidase were quantified in purified brush border fractions.

Main Results:

  • Misoprostol increased stomach and intestine relative weights, and mucosal weight in the duodenum and proximal jejunum.
  • Significant increases in sucrase, maltase, aminopeptidase, and alkaline phosphatase specific activities were observed throughout the small intestine.
  • Notably, sucrase activity increased by 62% in the proximal jejunum and 103% in the distal jejunum; alkaline phosphatase by 32% in duodenum and ileum; and aminopeptidase by 67% in the duodenum and 24% in the jejunum.

Conclusions:

  • Misoprostol administration significantly enhances brush border enzyme activity in the small intestine of growing rats.
  • These findings indicate that Misoprostol may improve the digestive functions of the small intestine.
  • Further research is warranted to elucidate the underlying cellular and molecular mechanisms responsible for these observed effects.

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