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Analysis of long-term viral expression in CEM cells persistently infected with non syncytium-inducing HIV-1 strains

J Yelle1, R Morisset, L Thibodeau

  • 1Centre de recherche en virologie, Institut Armand-Frappier, Laval, Québec, Canada.

Archives of Virology
|January 1, 1994
PubMed

Insights

This study reveals that human immunodeficiency virus type 1 (HIV-1) non-syncytium-inducing strains interact uniquely with CEM cell subpopulations. Long-term cultures show elimination of provirus-carrying cells, leaving only CD4-negative cells.

Area of Science:

  • Virology
  • Cell Biology
  • Immunology

Background:

  • Human immunodeficiency virus type 1 (HIV-1) infection establishes persistent reservoirs within host cells.
  • Understanding the interaction between HIV-1 strains and host cell subpopulations is crucial for developing effective therapeutic strategies.
  • CEM cells are a commonly used T-lymphoblastoid cell line for HIV-1 research.

Purpose of the Study:

  • To investigate the long-term effects of non-syncytium-inducing (NSI) HIV-1 strains on CEM cell subpopulations.
  • To characterize the cellular responses and viral dynamics in persistently infected CEM cultures.
  • To identify distinct cell types within the CEM line based on their interaction with NSI HIV-1.

Main Methods:

  • CEM cells were infected with three NSI HIV-1 strains.
  • Persistent cultures were maintained for several months.
  • Virus expression was analyzed using microscopy, immunofluorescence, reverse transcriptase assays, PCR, nucleic acid hybridization, and flow cytometry.
  • Superinfection resistance was assessed using the laboratory strain Lai.

Main Results:

  • NSI HIV-1 isolates induced minimal cytopathic effects and low percentages of virus-expressing cells (≤44%).
  • Persistent cultures showed resistance to superinfection, with transient antigen expression in one culture.
  • Long-term cultivation resulted in the elimination of antigen-positive and provirus-carrying cells, leaving only CD4-negative cells.
  • PCR analysis confirmed the progressive elimination of provirus-harboring cells.

Conclusions:

  • CEM cell populations contain distinct subpopulations with differential susceptibility to NSI HIV-1 infection.
  • CD4-positive cells are susceptible to rapid lysis, while others harbor latent provirus before potential activation and lysis.
  • Rare CD4-negative cells are selected for and survive NSI HIV-1 infection.
  • This study highlights the complex interplay between NSI HIV-1 and distinct CEM cell types, leading to eventual viral clearance from surviving cells.

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