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[Changes in rectal temperature during tympanoplasty under general anesthesia]

H Ohkawa1, T Iwakawa, N Ohtomo

  • 1Department of Anesthesiology, University of Hirosaki School of Medicine.

Masui. the Japanese Journal of Anesthesiology
|December 1, 1994
PubMed
Summary

Total intravenous anesthesia (DFK) significantly increased rectal temperature in tympanoplasty patients compared to enflurane or neuroleptic anesthesia. This suggests preserved thermoregulation may contribute to hyperthermia during DFK anesthesia.

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Area of Science:

  • Anesthesiology
  • Physiology
  • Surgical Patient Monitoring

Background:

  • Anesthesia can alter patient thermoregulation, potentially leading to intraoperative hyperthermia or hypothermia.
  • Understanding the impact of different anesthetic techniques on body temperature is crucial for patient safety during tympanoplasty.

Purpose of the Study:

  • To compare the effects of three general anesthesia types on rectal temperature during tympanoplasty.
  • To investigate the potential mechanisms behind observed temperature changes, particularly in total intravenous anesthesia.

Main Methods:

  • Thirty-five patients undergoing tympanoplasty were randomly assigned to three anesthesia groups: enflurane-N2O, neuroleptic anesthesia (droperidol, pentazocine-N2O), and total intravenous anesthesia (droperidol, fentanyl, ketamine - DFK).

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  • Rectal temperature was continuously monitored using an electric thermometer throughout the surgical procedure.

    • Main Results:

    • All three anesthetic groups exhibited an increase in rectal temperature.
    • The DFK group showed a significantly greater increase in rectal temperature compared to the enflurane and neuroleptic anesthesia groups.
    • No specific complications related to temperature changes were detailed in the abstract.

    Conclusions:

    • Total intravenous anesthesia with droperidol, fentanyl, and ketamine (DFK) is associated with a significant increase in rectal temperature during tympanoplasty.
    • The preserved hypothalamic thermoregulatory function and potential direct central nervous system stimulation may contribute to the observed hyperthermia in the DFK group.